This review examines NAD+ (nicotinamide adenine dinucleotide) as a critical cellular energy cofactor and its role in cardiovascular health. NAD+ levels naturally decline with age and disease, contributing to cardiovascular dysfunction. The review likely covers how NAD+ supplementation through precursors like NMN may help restore cellular energy metabolism and support heart health. This positions NAD+ enhancement as a therapeutic target for cardiovascular disease prevention and treatment.
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NAD+ decline with age links to cardiovascular dysfunction; NMN precursor support may be worth discussing with at-risk patients.